Obesity scapegoat wears thin
I always read advice columnists whenever I’m procrastinating at writing papers, and I came across this rather interesting letter to Dear Abby.
Despite a family history of high blood pressure, Dawn Edwards, a self-described “healthy, vibrant mother of one” and “active health-conscious vegetarian,” thought she was at her physical peak. So when she experienced nausea and lower back pain, she assumed she might be pregnant. Instead, she was shocked to learn she was in kidney failure. After 10 years of dialysis, Edwards received a transplant and now works to promote kidney disease awareness and education. Writes Edwards:
Many people don’t realize that high blood pressure and diabetes are the two leading causes of kidney disease. More than 26 million Americans have chronic kidney disease, and millions more are at risk. Screening for kidney function is not part of a routine physical examination, and kidney disease generally shows no symptoms — so if you have a family history of high blood pressure or diabetes, you are at risk.
Edwards’ letter doesn’t reveal her weight, but it is to be assumed by the descriptive characterization of her health, she is at least relatively thin. Of course, Edwards could be a proponent of Health at Every Size, in which it’s recognized that you can be healthy and active at any weight, but for the sake of this argument, let’s assume Edwards is among the majority of people who have been sold the special-interests-funded medical mantra that fat is unhealthy.
Which leads me to this: High blood pressure and especially the dread diabetes are two of the most common health-related disorders doctors and researchers like to use to fan the flames of obesity fear mongering. While there very may well exist a correlation, one’s family history and genetic predisposition remain the highest predictors for the great majority of illnesses and diseases commonly attributed up to obesity. By suffocating these disorders beneath a warm blanket of obesity, thin people like Edwards are led to develop a false sense of security in that because their BMI falls within that arbitrary and narrow range deemed “healthy,” their risk for developing such health-related problems is slight to none.
Take type 2 diabetes, for example, the most popular medical whipping boy of the anti-obesity establishment. To develop diabetes, you must first inherit the predisposition to diabetes and of the two types of diabetes, it is type 2 diabetes - the type most often blamed on obesity - that has the stronger genetic basis. In fact, family history is such an overwhelming and dominant factor in whether one develops diabetes that a recent study called for the formal addition of family history in public health strategies aimed at detecting and preventing the disease.
When a thin person develops diabetes or kidney failure, it’s most often attributed to a strong family history or to some freak anomaly of nature. Yet when a fat person with the same family history develops the same disorder, it’s automatically blamed on their fatness. Even more dangerous are those scary cases in which a person has a genuine disorder entirely unrelated to weight, but are dismissed by doctors who can’t see past weight to treat the real problem.
Environment is a trigger in many disorders, sure, but even people with extremely healthy lifestyles are destined to develop health problems if there exists a strong family history - just ask Kazz or Mike Benson. And I don’t dispute the role that nutrition or lack thereof plays in the development of some disorders, like diabetes. What I do dispute are the ungrounded assumptions that as a group, fat people eat poorly and engage in behaviors that are more likely to contribute to disorders like diabetes. In fact, studies have shown that fat people eat no differently than thin people, and in the case of diabetes, some studies claim that one’s diet is inconsequential in the development of the disease. I also question studies that purport the health hazards of obesity as an independent variable, especially when the data - which is often funded by groups with a financial vested interest in seeing a particular outcome - is questioned, refuted, reasserted and then disproven time and time again in what is known as the obesity paradox.
Poor nutrition and a sedentary lifestyle do cause health problems - in people of all sizes. So, too, does one’s genetic predisposition and family history play a strong, if not the strongest, role. By exaggerating and over-inflating the tenuous and often hypothetical influence of obesity in the development of illness and disease, the medical establishment is only hurting the very people they strive to help.
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